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MPTP-mediated ablation of enteric dopaminergic neurons, which reduced dopamine release in the gut, increased the production of inflammatory cytokines by invariant NK T-cells during hepatitis by preventing dopamine-induced repression of inflammatory signaling via the D1-like receptor-PKA pathway (Xue, Zhang et al., 2018)

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Notably, these interactions are speculated to be achieved through oxidative pathways and may occur independently of the HGH receptor in these models