S26A4 Polyclonal Antibody, 100ul Mammalian Expression Not detected in brain and

Not detected in brain and skeletal muscle

May play a role in the trafficking of activated/effector T-lymphocytes to inflammatory sites and other aspects of activated T-lymphocyte physiology

with a role in targeting this protein to chromatin during anaphase

2001 [PubMed 11689425])

Several human genetic disorders are caused by aberrations in human homeobox genes

S26A4 Polyclonal Antibody, 100ul Mammalian Expression Not detected in brain andMutations in this gene are associated with Pendred syndrome, the most common form of syndromic deafness, an autosomal recessive disease. It is highly homologous to the SLC26A3 gene; they have similar genomic structures and this gene is located 3' of the SLC26A3 gene. The encoded protein has homology to sulfate transporters.